. The Glycemic Index offers an exciting, and scientifically valid, means of determining the types of carbohydrates we eat. I-t measures and costs the way these carbohydrates enter the body. And in doing so offers a means of approaching meals so that it might be both nutritionally valid, and keep food cravings away. Clicking enstep.com seemingly provides warnings you might tell your sister. This obviously is one way of preventing overeating. And its an effective way for parents to try and control their kids' desire to eat un-healthy food between meals.

The Glycemic Index rates carbs as having the high, low or moderate glycemic index. And the concept would be to eat more foods which have the low or medium glycemic index, and less with a high one. Reduced glycemic index foods enter the bloodstream more slowly, and therefore do not increase blood sugar like high glycemic index foods.

Therefore, what's a carbohydrate? All sugars, or foods that are separated into sugar, are carbohydrates. Including standard sugar, glucose (often used in sports drinks), fructose, (in good fresh fruit), lactose, (found in milk and similar services and products like yoghurt), maltose, (found in malt that will be often used to taste cereals), all types of starches, from potatoes to noodles and rice, and legumes, such as for example peas and peas (though these also contain some protein).

Fruit is considered to possess a low-gi (not fruit juice however). Interestingly though, recent re-search has found what they think is a link between fructose and obesity. But, the type of fructose examined was in corn syrup, which is a refined and concentrated form of fructose. It also doesn't have the fiber, anti-oxidants and other phytochemicals that fresh fruit does. This was also research done within an animal model, therefore it may possibly not be appropriate for humans. Researchers from the University of Florida discovered that fructose may make people believe they're hungrier than they should really be. And when these experts abandoned just how fructose was metabolized, the mice they were working with didn't put on weight, although they still ate fructose.

This is simply not the very first study that's suggested fructose might be linked to a trend to put on weight, moreso than other forms of food. Research at the University of Cincinnati discovered that eating fructose (high fructose corn syrup), generated greater fat storage. They say although again, it is unclear if this can be mitigated by probably the lower concentration of fructose in good fresh fruit as compared to the corn syrup used in the study, that the body functions fructose differently to other forms of sugars.

The investigation in the University of Florida discovered that there were higher degrees of uric acid in the body after eating or drinking fructose. Insulin is affected by this spike in uric acid, by preventing it. Insulin regulates the way our cells shop and use fat. If uric acid levels are elevated a whole lot, then apparent symptoms of metabolic syndrome may develop. These signs include high blood pressure, high cholesterol levels, as well as developing a lot of weight. What is of possible problem to people is that fructose is employed in a lot of soda products, so it is going to be quite easy to generally raise uric acid levels in the body if you drink a lot of soda. Metabolic syndrome can also be a precursor of type 2 diabetes.

Signs of metabolic syndrome include fat around the stomach, such that the waist seems as huge as the hips or larger. There are generally lower portions of the good type of cholesterol in the blood, and high levels of triglycerides which will make the blood 'sticky.' Metabolic syndrome is associated with the way the body responds to insulin, so that you'll find higher degrees of sugar in the body. Most of these things may be tested by medical practioners.

References:

1. http://www.nutraingredients.com/news/ng.asp?n=64395&m=1NIED08&c=qgtqmovbyiaxdub

2. Australian Healthy Food, December 2005

3. Nature and Health, Oct/Nov 2005.

 
is_fructose_the_cause_of_obesity.txt · Dernière modification: 2013/10/29 19:12 (édition externe)
 
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